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CHARTS
COMPLICATIONS
• Contractile dysfunction
• Right ventricular infarction
• Papillary dysfunction
• Myocardial rupture
• Arrhythmias
• Pericarditis
• Chamber dilation
• Mural thrombus
• Ventricular aneurysm
• Progressive heart failure
PNEUMONIA
• Bacterial pneumonia has two patterns of anatomic distribution:
lobular bronchopneumonia and lobar pneumonia
• Patchy consolidation of the lung is the dominant characteristic of
bronchopneumonia.
• The consolidation may be confined to one lobe but is
more often multilobar and frequently bilateral and basal
because of the tendency of secretions to gravitate to the
lower lobes.
PEPTIC ULCER
• Ulcer: Breach in muscularis mucosa of GI tract
• Erosion is more superficial breach
• Ulcers begin as erosions, but not all erosions progress to
ulcers
• Peptic ulcer:
• Chronic, usually solitary, due to acid-peptic juices
ACUTE GASTRITIS
• Heavy smoking
• Excessive alcohol
• Excessive NSAID-Aspirin,
Ibuprofen
• Uremia
• Ischemia and shock
• Stress( Major surgery,
burns, severe infections)
CHRONIC GASTRITIS
• Drugs- NSAID
• H.pylori
• Alcohol and smoking
• Radiation
• Pernicious anemia
GROSS
• 90% < 4 cm, 50% < 2cm, clean base (due to peptic
enzymes), surrounded by erythematous mucosa, may see
blood vessel in ulcer base
• Usually sharply punched out defect with straight walls,
NO heaped up margins
• Size doesn’t predict malignancy
H.PYLORI
HISTOLOGY H.PYLORI
• superficial band of predominately chronic inflammation,
H. PYLORI
AMYLOIDOSIS
• Amyloidosis is a condition associated with a number of
inherited and inflammatory disorders in which
extracellular deposits of fibrillar proteins are responsible
for tissue damage and functional compromise
• Autopsy specimen of a patient who died due to MI was
stained with triphenyltetrazolium chloride dye. Colour of
normal part of heart will be:
1. Blue
2. White
3. Red
4. Dark brown
• Autopsy specimen of a patient who died due to MI was
stained with triphenyltetrazolium chloride dye. Colour of
normal part of heart will be:
1. Blue
2. White
3. Red
4. Dark brown
• Earliest light microscopic change in MI
1. Waviness of fibres
2. Neutrophilic infiltration
3. Coagulation necrosis
4. Contraction band necrosis
• Earliest light microscopic change in MI
• Waviness of fibres
• Neutrophilic infiltration
• Coagulation necrosis
• Contraction band necrosis
• Type of necrosis seen in MI
1. Caseous
2. Coagulative
3. Liquefactive
4. Fibrinoid
• Type of necrosis seen in MI
1. Caseous
2. Coagulative
3. Liquefactive
4. Fibrinoid
• Most common site of peptic (gastric) ulcer
1. Upper third of lesser curvature
2. Greater curvature
3. Pyloric antrum
4. Lesser curvature
• Most common site of peptic (gastric) ulcer
1. Upper third of lesser curvature
2. Greater curvature
3. Pyloric antrum
4. Lesser curvature
• Terminal stage of pneumonia is
1. Congestion
2. Red hepatization
3. Grey hepatization
4. Resolution
• Terminal stage of pneumonia is
1. Congestion
2. Red hepatization
3. Grey hepatization
4. Resolution
• In stage of grey hepatization
1. WBC fill the alveoli
2. RBC fill the alveoli
3. Organisms fill the alveoli
4. Accumulation of fibrin
• In stage of grey hepatization
1. WBC fill the alveoli
2. RBC fill the alveoli
3. Organisms fill the alveoli
4. Accumulation of fibrin
• Most widely used stain for amyloidosis
1. Oil red O
2. Congo red
3. PAS
4. Thioflavin T
• Most widely used stain for amyloidosis
1. Oil red O
2. Congo red
3. PAS
4. Thioflavin T
• Skin biopsy in amyloidosis
1. Kidney
2. Lip
3. Abdominal fat
4. rectum
• Skin biopsy in amyloidosis
1. Kidney
2. Lip
3. Abdominal fat
4. Rectum
• Best investigation for amyloidosis
1. Rectal bx
2. CT scan
3. MRI
4. Colonoscopy
• Best investigation for amyloidosis
1. Rectal bx
2. CT scan
3. MRI
4. Colonoscopy

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CHARTS.pptx

  • 2.
  • 3.
  • 4.
  • 5.
  • 6. COMPLICATIONS • Contractile dysfunction • Right ventricular infarction • Papillary dysfunction • Myocardial rupture • Arrhythmias • Pericarditis • Chamber dilation • Mural thrombus • Ventricular aneurysm • Progressive heart failure
  • 7.
  • 8. PNEUMONIA • Bacterial pneumonia has two patterns of anatomic distribution: lobular bronchopneumonia and lobar pneumonia • Patchy consolidation of the lung is the dominant characteristic of bronchopneumonia.
  • 9.
  • 10.
  • 11. • The consolidation may be confined to one lobe but is more often multilobar and frequently bilateral and basal because of the tendency of secretions to gravitate to the lower lobes.
  • 12.
  • 13.
  • 14.
  • 15. PEPTIC ULCER • Ulcer: Breach in muscularis mucosa of GI tract • Erosion is more superficial breach • Ulcers begin as erosions, but not all erosions progress to ulcers • Peptic ulcer: • Chronic, usually solitary, due to acid-peptic juices
  • 16. ACUTE GASTRITIS • Heavy smoking • Excessive alcohol • Excessive NSAID-Aspirin, Ibuprofen • Uremia • Ischemia and shock • Stress( Major surgery, burns, severe infections) CHRONIC GASTRITIS • Drugs- NSAID • H.pylori • Alcohol and smoking • Radiation • Pernicious anemia
  • 17. GROSS • 90% < 4 cm, 50% < 2cm, clean base (due to peptic enzymes), surrounded by erythematous mucosa, may see blood vessel in ulcer base • Usually sharply punched out defect with straight walls, NO heaped up margins • Size doesn’t predict malignancy
  • 19. HISTOLOGY H.PYLORI • superficial band of predominately chronic inflammation,
  • 21.
  • 22. AMYLOIDOSIS • Amyloidosis is a condition associated with a number of inherited and inflammatory disorders in which extracellular deposits of fibrillar proteins are responsible for tissue damage and functional compromise
  • 23.
  • 24.
  • 25. • Autopsy specimen of a patient who died due to MI was stained with triphenyltetrazolium chloride dye. Colour of normal part of heart will be: 1. Blue 2. White 3. Red 4. Dark brown
  • 26. • Autopsy specimen of a patient who died due to MI was stained with triphenyltetrazolium chloride dye. Colour of normal part of heart will be: 1. Blue 2. White 3. Red 4. Dark brown
  • 27. • Earliest light microscopic change in MI 1. Waviness of fibres 2. Neutrophilic infiltration 3. Coagulation necrosis 4. Contraction band necrosis
  • 28. • Earliest light microscopic change in MI • Waviness of fibres • Neutrophilic infiltration • Coagulation necrosis • Contraction band necrosis
  • 29. • Type of necrosis seen in MI 1. Caseous 2. Coagulative 3. Liquefactive 4. Fibrinoid
  • 30. • Type of necrosis seen in MI 1. Caseous 2. Coagulative 3. Liquefactive 4. Fibrinoid
  • 31. • Most common site of peptic (gastric) ulcer 1. Upper third of lesser curvature 2. Greater curvature 3. Pyloric antrum 4. Lesser curvature
  • 32. • Most common site of peptic (gastric) ulcer 1. Upper third of lesser curvature 2. Greater curvature 3. Pyloric antrum 4. Lesser curvature
  • 33. • Terminal stage of pneumonia is 1. Congestion 2. Red hepatization 3. Grey hepatization 4. Resolution
  • 34. • Terminal stage of pneumonia is 1. Congestion 2. Red hepatization 3. Grey hepatization 4. Resolution
  • 35. • In stage of grey hepatization 1. WBC fill the alveoli 2. RBC fill the alveoli 3. Organisms fill the alveoli 4. Accumulation of fibrin
  • 36. • In stage of grey hepatization 1. WBC fill the alveoli 2. RBC fill the alveoli 3. Organisms fill the alveoli 4. Accumulation of fibrin
  • 37. • Most widely used stain for amyloidosis 1. Oil red O 2. Congo red 3. PAS 4. Thioflavin T
  • 38. • Most widely used stain for amyloidosis 1. Oil red O 2. Congo red 3. PAS 4. Thioflavin T
  • 39. • Skin biopsy in amyloidosis 1. Kidney 2. Lip 3. Abdominal fat 4. rectum
  • 40. • Skin biopsy in amyloidosis 1. Kidney 2. Lip 3. Abdominal fat 4. Rectum
  • 41. • Best investigation for amyloidosis 1. Rectal bx 2. CT scan 3. MRI 4. Colonoscopy
  • 42. • Best investigation for amyloidosis 1. Rectal bx 2. CT scan 3. MRI 4. Colonoscopy