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• The term hypermetropia is derived from
  hyper meaning “In excess” met meaning
   “measure” & opia meaning “of the eye”.

• Also called hyperopia / longsightedness

• First suggested in 1755 by KASTNER
DEFINITION
• It is the refractive state of eye where in
  parallel rays of light coming from infinity
  are focused behind the sentient layer of
  retina with accommodation being at rest
• The posterior focal point is behind the
  retina which receives a blurred image
ETIOLOGY
1) AXIAL
• Most common
• Total refractive power of eye is normal
• Axial shortening of eyeball
• 1mm short- 3 D of HM
• Physiologically more than 6D HM are
  uncommon
• At birth +2.5 – 3 D of HM (physiologically)
• Pathologically seen in cases like orbital tumour,
  inflammatory mass , oedema, coloboma and
  microphthalmos.
2) CURVATURAL
• Flattening of cornea, lens or both
• 1mm increase in Radius of curvature-
  RESULTS IN 6D of HM
• Never exceed 6D HM physiologically
• Congenitally flattened (cornea plana)
• Result (trauma and disease )
3) INDEX
• Change in refractive index with age
• Physiologically in old age
• Pathologically in diabetics under treatment
4)POSITIONAL
• Posteriorly placed crystalline lens
• Occurs as congenital anomaly
• Result of trauma or disease
5)ABSENCE OF LENS
• Seen in aphakia
CLINICAL TYPES

• SIMPLE HYPERMETROPIA,
• PATHOLOGICAL
• FUNCTIONAL HYPEROPIA
SIMPLE HYPERMETROPIA
• Commonest form
• Results from normal biological variations
  in the development of eyeball
• Include axial and curvatural HM
• May be hereditary
PATHOLOGICAL HYPERMETROPIA
• Anomalies lie outside the limits of biological
  variation
• Acquired hypermetropia
  – Decrease curvature of outer lens fibers in old
    age
  – Cortical sclerosis


• Positional hypermetropia
• Aphakia
• Consecutive hypermetropia
FUNCTIONAL HYPERMETROPIA
• Results from paralysis of accommodation

• Seen in patients with 3rd nerve paralysis &
  internal ophthalmoplegia
OPTICAL CONDITION
• Parallel rays focus behind retina
• Diffusion circles produce blurred & indistinct
  images
• Retina is nearer to nodal point
• Image is smaller than in emmetropic
• Rays diverge from retina
• Formation of clear image is possible only when
  converging power of eye is increased
NOMENCLATURE



TOTAL HYPERMETROPIA=
LATENT + MANIFEST
        (facultative + absolute)
TOTAL HYPERMETROPIA
• It is the total amount of refractive
  error,estimated after complete cycloplegia
  with atropine

• Divided into latent & manifest
LATENT HYPERMETROPIA
• Corrected by inherent tone of ciliary
  muscle
• Usually about 1D
• High in children
• Decreases with age
• Revealed after abolishing tone of ciliary
  muscle with atropine
MANIFEST HYPERMETROPIA
• Remaining part of total hypermetropia
• Correct by accommodation and convex lens
• Measure by add strongest lens with max. vision
• Consists of facultative & absolute
FACULTATIVE HYPERMETROPIA
• Corrected by patients accommodative effort
ABSOLUTE HYPERMETROPIA
• Residual part not corrected by patients
  accommodative effort
  Absolute hypermetropia can be measured by the
  weakest convex lens with which maximum visual
  acuity
MANIFEST HYPERMETROPIA
            CONT…
• Manifest HM – absolute HM = Facultative HM
(Strongest lens) – (weakest lens)
• Total HM – Manifest HM = Latent HM
NORMAL AGE VARIATION
 At birth +2+3D HM
• Slightly increase in one year of life,
• Gradually diminished untill by the age 5-10 years
 In old age after 50 year again tendency to HM

   Ton of ciliary muscle decreases
   Accommodative power decreases
   Some amount of latent HM become manifest
   More amount of facultative HM become absolute
   Practically after 65 year all of it become absolute
SYMPTOMS
• Principal symptom is blurring of vision for close
  work
• Symptoms vary depending upon age of patient &
  degree of refractive error
ASYMPTOMATIC
• small error produces no symptoms
• Corrected by accommodation of patient
ASTHENOPIA
• Refractive error are fully corrected by
  accommodative effort
• Thus vision is normal
• Sustained accommodation produces symptoms
• Asthenopia increases as day progresses
• Increased after prolonged near work
SYMPTOMS
            Tiredness
            Frontal or fronto temporal headache
            Watering
            Mild photophobia
DEFECTIVE VISION WITH ASTHENOPIA

• Not corrected by accommodation
• Defective vision for near more than
  distance
• Asthenopia due to sustained
  accommodation
• Refractive error more(>4D)
DEFECTIVE VISION ONLY
• Refractive vision more than 4D
• Adults usually do not accommodate
• Marked defective vision for near and
  distance
SIGNS
• VISUAL ACUITY : Defective
• EYEBALL: small or normal in size
• CORNEA : may be smaller than normal.
  There can be CORNEA PLANA
• ANTERIOR CHAMBER : may be shallow
• LENS: could be dislocated backwards
• A Scan ultrasonography (biometry) reveal
  short axial length
FUNDUS:
B) DISC: Dark reddish color, irregular
   margins ,confused with Papillitis so
   termed as PSEUDO-PAPILLITIS
C) MACULA: Situated further from the disc
   than usual, large positive angle alpha,
   apparent divergent squint
D) BLOOD VESSELS: Show undue
   tortuosity & abnormal branchings
E) BACKGROUND: SHOT- SILK RETINA
COMPLICATION
• Recurrent styes m blepharitis or chalazia
• Accommodative convergent squint
• Amblyopia
  – Anisometropic
  – Stravismic
  – Uncorrective bilateral high hypermetropia
• Predisposition to develop primary narrow
  angle glaucomas
   Care should be taken while instilling
 mydriatics
TREATMENT

BASIS FOR TREATMENT
• No Treatment
Error is small
Asymptomatic
Visual acuity normal
No muscular imbalance
Young children(<6 or 7yrs)
 Some degree of hypermetropia is physiological so
  no correction
 Treatment required if error is high or strabismus is
  present
 working in school small error may require
  correction
 In children error tends normally to diminish with
  growth so refraction should be carried out every six
  month and if necessary the correction should be
  reduced, ortherwise a lens which is overcorrecting
  their error may induce an artificial myopia
 No deduction of tonus allowance in strabismus
ADULTS
If symptoms of eye-strain are marked,we
 correct as much of the total
 hypermetropia as possible,trying as far as
 we can to relieve the accommodation
When there is spasm of accommodation
 we correct the whole of the error
Some patients with hypermetropia do not
 initially tolerate the full correction
 indicated by manifest refraction so we
 undercorrect them
Exophoria hyperopia should be under
 correct by 1 to 2D
Patients with absolute hypermetropia
 are more likely to accept nearly the
 full correction because they typically
 experience immediate improvement
 in visual acuity
In pathological hypermetropia the
 underlying cause rather than the
 hypermetropia is chief concern
MODE OF TREATMENT
• SPECTACLES
                 OPTICAL TREATMENT
• CONTACT LENS

• SURGICAL
SPECTACLES
Basic principle
  Prescribe convex lenses(Plus lenses)         so
  that rays are brought to focus on the retina
Advantages
• Comfortable
• Easier method
• Less expensive
• Safe idea
CONTACT LENS
ADVANTAGES
Cosmetically good

Increased field of view

Less magnification

Elimination of aberrations & prismatic effect
REFRACTIVE SURGERY
• Refractive surgery is not as effective as in
  myopia
TYPES:

(1)HEXAGONAL KERATOTOMY(HK)
• Low to moderate degrees of hypermetropia
• Its risk /benefit ratio is not low enough to warrant
  its continued use
LASER THERMAL
       KERATOPLASTY(LTK)
• Procedure done using laser energy to
  heat the cornea (contraction of collagen)
  and increase its curvature
• Central heating of cornea results in central
  corneal flattening thereby resulting in
  hyperopic shift
PHOTOREFRACTIVE
       KERATECTOMY(PRK)
• Direct laser ablation of corneal stroma
  after removal of corneal epithelium
  mechanically
• Done using EXCIMER LASER
LASER IN SITU
     KERATOMILEUSIS(LASIK)
• Anterior flap of cornea lifted with keratome
  and excimer laser is used to sculpt the
  stromal bed to change the refractive error
  of eye

• It can correct up to 4D of hypermetropia
  and 8D of astigmatism
PHAKIC IOL AND CLEAR LENS
        EXTRACTION
• Done by Phaco technique

• Clear lens extraction with the implantation of an
  IOL-----Preferably foldable IOL or a Piggyback
  IOL is implanted
VISUAL HYGIENE
• While reading or doing intensive near work take
  a break about every 30 min
• When reading maintain proper distance that is
  the book should be at least as far from your
  eyes as your elbow when you make a fist and
  hold it against your nose
• Sufficient Illumination
• Place a limit spent watching television &
  watching videogames
• Sit 5-6 feet away from the television
• Appropriate optical correction almost
  always leads to clear and comfortable
  single binocular vision
• Younger children who have significant
  hyperopia associated with amblyopia,
  strabismus,or     anisometropia     require
  treatment, starting as early as 3-6 months
  of age
CONCLUSION
• Hyperopia is a common refractive disorder that
  has been overshadowed by myopia in public
  perception,vision research & the scientific
  literature
• Although uncorrected myopia has a greater
  adverse effect on visual acuity than uncorrected
  hyperopia,the close association between
  hyperopia,amblyopia & strabismus,especially in
  children,makes hyperopia a greater risk factor
  for more permanent vision loss than myopia
• The early diagnosis & treatment of
  significant hyperopia & its consequences
  can prevent a significant amount of visual
  disability in the general population
For Further Queries Contact :
Ms. Priyanka Singh
Head – Optometry Service
Email – optometry@venueyeinstitute.org

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Hypermetropia

  • 1.
  • 2. • The term hypermetropia is derived from hyper meaning “In excess” met meaning “measure” & opia meaning “of the eye”. • Also called hyperopia / longsightedness • First suggested in 1755 by KASTNER
  • 3. DEFINITION • It is the refractive state of eye where in parallel rays of light coming from infinity are focused behind the sentient layer of retina with accommodation being at rest • The posterior focal point is behind the retina which receives a blurred image
  • 4.
  • 5. ETIOLOGY 1) AXIAL • Most common • Total refractive power of eye is normal • Axial shortening of eyeball • 1mm short- 3 D of HM • Physiologically more than 6D HM are uncommon • At birth +2.5 – 3 D of HM (physiologically) • Pathologically seen in cases like orbital tumour, inflammatory mass , oedema, coloboma and microphthalmos.
  • 6. 2) CURVATURAL • Flattening of cornea, lens or both • 1mm increase in Radius of curvature- RESULTS IN 6D of HM • Never exceed 6D HM physiologically • Congenitally flattened (cornea plana) • Result (trauma and disease ) 3) INDEX • Change in refractive index with age • Physiologically in old age • Pathologically in diabetics under treatment
  • 7. 4)POSITIONAL • Posteriorly placed crystalline lens • Occurs as congenital anomaly • Result of trauma or disease 5)ABSENCE OF LENS • Seen in aphakia
  • 8. CLINICAL TYPES • SIMPLE HYPERMETROPIA, • PATHOLOGICAL • FUNCTIONAL HYPEROPIA
  • 9. SIMPLE HYPERMETROPIA • Commonest form • Results from normal biological variations in the development of eyeball • Include axial and curvatural HM • May be hereditary
  • 10. PATHOLOGICAL HYPERMETROPIA • Anomalies lie outside the limits of biological variation • Acquired hypermetropia – Decrease curvature of outer lens fibers in old age – Cortical sclerosis • Positional hypermetropia • Aphakia • Consecutive hypermetropia
  • 11. FUNCTIONAL HYPERMETROPIA • Results from paralysis of accommodation • Seen in patients with 3rd nerve paralysis & internal ophthalmoplegia
  • 12. OPTICAL CONDITION • Parallel rays focus behind retina • Diffusion circles produce blurred & indistinct images • Retina is nearer to nodal point • Image is smaller than in emmetropic • Rays diverge from retina • Formation of clear image is possible only when converging power of eye is increased
  • 13.
  • 14.
  • 15. NOMENCLATURE TOTAL HYPERMETROPIA= LATENT + MANIFEST (facultative + absolute)
  • 16. TOTAL HYPERMETROPIA • It is the total amount of refractive error,estimated after complete cycloplegia with atropine • Divided into latent & manifest
  • 17. LATENT HYPERMETROPIA • Corrected by inherent tone of ciliary muscle • Usually about 1D • High in children • Decreases with age • Revealed after abolishing tone of ciliary muscle with atropine
  • 18. MANIFEST HYPERMETROPIA • Remaining part of total hypermetropia • Correct by accommodation and convex lens • Measure by add strongest lens with max. vision • Consists of facultative & absolute FACULTATIVE HYPERMETROPIA • Corrected by patients accommodative effort ABSOLUTE HYPERMETROPIA • Residual part not corrected by patients accommodative effort Absolute hypermetropia can be measured by the weakest convex lens with which maximum visual acuity
  • 19. MANIFEST HYPERMETROPIA CONT… • Manifest HM – absolute HM = Facultative HM (Strongest lens) – (weakest lens) • Total HM – Manifest HM = Latent HM
  • 20. NORMAL AGE VARIATION  At birth +2+3D HM • Slightly increase in one year of life, • Gradually diminished untill by the age 5-10 years  In old age after 50 year again tendency to HM  Ton of ciliary muscle decreases  Accommodative power decreases  Some amount of latent HM become manifest  More amount of facultative HM become absolute  Practically after 65 year all of it become absolute
  • 21. SYMPTOMS • Principal symptom is blurring of vision for close work • Symptoms vary depending upon age of patient & degree of refractive error ASYMPTOMATIC • small error produces no symptoms • Corrected by accommodation of patient
  • 22. ASTHENOPIA • Refractive error are fully corrected by accommodative effort • Thus vision is normal • Sustained accommodation produces symptoms • Asthenopia increases as day progresses • Increased after prolonged near work SYMPTOMS Tiredness Frontal or fronto temporal headache Watering Mild photophobia
  • 23. DEFECTIVE VISION WITH ASTHENOPIA • Not corrected by accommodation • Defective vision for near more than distance • Asthenopia due to sustained accommodation • Refractive error more(>4D)
  • 24. DEFECTIVE VISION ONLY • Refractive vision more than 4D • Adults usually do not accommodate • Marked defective vision for near and distance
  • 25. SIGNS • VISUAL ACUITY : Defective • EYEBALL: small or normal in size • CORNEA : may be smaller than normal. There can be CORNEA PLANA • ANTERIOR CHAMBER : may be shallow • LENS: could be dislocated backwards • A Scan ultrasonography (biometry) reveal short axial length
  • 26. FUNDUS: B) DISC: Dark reddish color, irregular margins ,confused with Papillitis so termed as PSEUDO-PAPILLITIS C) MACULA: Situated further from the disc than usual, large positive angle alpha, apparent divergent squint D) BLOOD VESSELS: Show undue tortuosity & abnormal branchings E) BACKGROUND: SHOT- SILK RETINA
  • 27. COMPLICATION • Recurrent styes m blepharitis or chalazia • Accommodative convergent squint • Amblyopia – Anisometropic – Stravismic – Uncorrective bilateral high hypermetropia • Predisposition to develop primary narrow angle glaucomas Care should be taken while instilling mydriatics
  • 28. TREATMENT BASIS FOR TREATMENT • No Treatment Error is small Asymptomatic Visual acuity normal No muscular imbalance
  • 29. Young children(<6 or 7yrs)  Some degree of hypermetropia is physiological so no correction  Treatment required if error is high or strabismus is present  working in school small error may require correction  In children error tends normally to diminish with growth so refraction should be carried out every six month and if necessary the correction should be reduced, ortherwise a lens which is overcorrecting their error may induce an artificial myopia  No deduction of tonus allowance in strabismus
  • 30. ADULTS If symptoms of eye-strain are marked,we correct as much of the total hypermetropia as possible,trying as far as we can to relieve the accommodation When there is spasm of accommodation we correct the whole of the error Some patients with hypermetropia do not initially tolerate the full correction indicated by manifest refraction so we undercorrect them Exophoria hyperopia should be under correct by 1 to 2D
  • 31. Patients with absolute hypermetropia are more likely to accept nearly the full correction because they typically experience immediate improvement in visual acuity In pathological hypermetropia the underlying cause rather than the hypermetropia is chief concern
  • 32. MODE OF TREATMENT • SPECTACLES OPTICAL TREATMENT • CONTACT LENS • SURGICAL
  • 33. SPECTACLES Basic principle Prescribe convex lenses(Plus lenses) so that rays are brought to focus on the retina Advantages • Comfortable • Easier method • Less expensive • Safe idea
  • 34.
  • 35. CONTACT LENS ADVANTAGES Cosmetically good Increased field of view Less magnification Elimination of aberrations & prismatic effect
  • 36. REFRACTIVE SURGERY • Refractive surgery is not as effective as in myopia TYPES: (1)HEXAGONAL KERATOTOMY(HK) • Low to moderate degrees of hypermetropia • Its risk /benefit ratio is not low enough to warrant its continued use
  • 37.
  • 38. LASER THERMAL KERATOPLASTY(LTK) • Procedure done using laser energy to heat the cornea (contraction of collagen) and increase its curvature • Central heating of cornea results in central corneal flattening thereby resulting in hyperopic shift
  • 39. PHOTOREFRACTIVE KERATECTOMY(PRK) • Direct laser ablation of corneal stroma after removal of corneal epithelium mechanically • Done using EXCIMER LASER
  • 40. LASER IN SITU KERATOMILEUSIS(LASIK) • Anterior flap of cornea lifted with keratome and excimer laser is used to sculpt the stromal bed to change the refractive error of eye • It can correct up to 4D of hypermetropia and 8D of astigmatism
  • 41. PHAKIC IOL AND CLEAR LENS EXTRACTION • Done by Phaco technique • Clear lens extraction with the implantation of an IOL-----Preferably foldable IOL or a Piggyback IOL is implanted
  • 42. VISUAL HYGIENE • While reading or doing intensive near work take a break about every 30 min • When reading maintain proper distance that is the book should be at least as far from your eyes as your elbow when you make a fist and hold it against your nose • Sufficient Illumination • Place a limit spent watching television & watching videogames • Sit 5-6 feet away from the television
  • 43. • Appropriate optical correction almost always leads to clear and comfortable single binocular vision • Younger children who have significant hyperopia associated with amblyopia, strabismus,or anisometropia require treatment, starting as early as 3-6 months of age
  • 44. CONCLUSION • Hyperopia is a common refractive disorder that has been overshadowed by myopia in public perception,vision research & the scientific literature • Although uncorrected myopia has a greater adverse effect on visual acuity than uncorrected hyperopia,the close association between hyperopia,amblyopia & strabismus,especially in children,makes hyperopia a greater risk factor for more permanent vision loss than myopia
  • 45. • The early diagnosis & treatment of significant hyperopia & its consequences can prevent a significant amount of visual disability in the general population
  • 46. For Further Queries Contact : Ms. Priyanka Singh Head – Optometry Service Email – optometry@venueyeinstitute.org