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DROWNING
AND
NEAR DROWNING
PRESENTED BY:-
Dr.K.AGASYA RAJ
WHAT IS DROWNING?
“The process of experiencing
respiratory impairment from
submersion/immersion in liquid ”
It is a form of asphyxia.
Caused due to aspiration of fluids
Epidemiology
Incidence of drowning- 2.5 lakhs to 3.5
lakhs every year
India & China account for 43% deaths.
Males>Females
1-4 yrs & 15-19 yrs(M.C)
Drowning second most common
accidental cause of death in children
Causes of Drowning
CHILDREN YOUNGER THAN 1 YR :
◩‱ BATHTUB(71%)
◩ ‱ HOUSEHOLD BUCKETS(16%)
CHILDREN 1-4 YR :
◩ ‱ POOL
◩ ‱ IRRIGATION DITCHES
◩ ‱ NEARBY PONDS & RIVERS.
ï‚—â€ą SCHOOL AGE CHILDREN
◩‱ SWIMMING OR BOATING ACTIVITIES.
◩‱ NATURAL WATER RESERVOIRS :
LAKES, PONDS, RIVERS, CANALS.
Causes of Drowning
ADOLESCENT
l M : F = 10 : 1.
◩(LIKELY DUE TO GREATER RISK
TAKING & ALCOHOL USE.)
◩70 % DEATHS DUE TO DROWNING IN
NATURAL WATER RESERVOIRS.
ï‚—â€ą UNDERLYING CONDITIONS
◩ EPILEPSY
◩ VENTRICULAR ARRHYTHMIAS
◩ ALCOHOL USE
◩ WATER SPORTS & RECREATIONAL
ACTIVITIES
Classification of drowning.
According to type of water
◩Fresh-water drowning: Lakes,pools,rivers
◩Salt-water drowning: seas (about 3%
saline)
ï‚—â€ą According to water temperature
◩Warm-water drowning: temperatures of >
20° C
lCold-water drowning: temperatures of
<20° C
◩Very-cold-water drowning: temperatures of
<5ÂșC
TYPES OF DROWNING
FOUR TYPES:-
1.WET DROWNING
2.DRY DROWNING
3.SECONDARY/NEAR DROWNING
4.IMMERSION SYNDROME
WET DROWNING
Water is inhaled into lungs
Victim has severe chest pain
On resuscitation: no pleasant
recollections
Death occurs due to Cardiac arrest or
ventricular fibrillation
Dry drowning
Water does not enter lungs
On resuscitation : panoramic views of
past life.
Death occurs by immediate sustained
laryngeal spasm due to inrush of water
into nasopharynx and larynx
Secondary drowning (near
drowning)
Refers to a submerged victim who is
resuscitated and survives for 24 hours
Death occurs ( from 1/2 h to several
days) by cerebral anoxia & irreversible
brain damage
Immersion Syndrome
Death occurs by cardiac arrest caused
by vagal inhibition (cold water
stimulating nerve endings & water
striking epigastrium & Alcohol induce
such effect)
Mostly seen in suicide cases.
Stages of drowning
Stage of surprise(5-10 secs)
Stage of 1st resp.arrest.(1-2 min)
Stage of deep respiration(1 min)
Stage of 2nd resp.arrest (1 min)
Stage of terminal gasp.
lFRESH WATER
DROWNING
Water absorbed into
circulation
Surfactant washout
Alveolar cell damage
Chemical pneumonitis,
pulmonary edema
Hypervolemia
Hyponatremia
Hemodilution
Hemolysis
Hyperkalemia
lSALT WATER
DROWNING
Hypertonic
Protein rich effusion into
alveoli
Surfactant damage,
alveolar basement
membrane damage
Alveolar cell damage
Chemical pneumonitis,
pulmonary edema
Hypovolemia
Hypernatremia
Hemoconcentration
Hypotonic
Cause of death
Asphyxia: due to airway obstruction
Hypothermia
Ventricular fibrillation: due to anoxia &
disturbed sodium-potassium ratio
Laryngeal spasm
Vagal inhibition: in icy water due to high
emotion & unexpected immersion
Exhaustion
Injury
SYMPTOMS AND SIGNS
75% of kids who develop symptoms do
so within 7 hours of event
Coma to agitated alertness
Cyanosis, coughing, and the production
of frothy white sputum
Tachypnea, tachycardia
Low-grade fever
Rales, rhonchi & less often wheezes
Signs of associated trauma to the head
and neck should be sought
History is the most important
EFFECT ON VARIOUS
SYSTEMS
LUNG :
◩ARDS.
◩PULMONARY OEDEMA
HEART :
◩MYOCARDIAL DYSFUNCTION.
◩ARTERIAL HYPOTENSION.
◩DECREASED CARDIAC OUTPUT.
◩ARRHYTHMIAS & CARDIAC INFARCTION.
 RENAL :
◩ACUTE TUBULAR NECROSIS
◩CORTICAL NECROSIS
◩RENAL FAILURE.
VASCULAR ENDOTHELIAL INJURY MAY INITIATE
◩DIC,
◩HEMOLYSIS,
◩THROMBOCYTOPENIA.
 GASTROINTESTINAL DAMAGE :
◩BLOODY DIARRHEA WITH MUCOSAL
SLOUGHING.
◩SERUM LEVELS OF HEPATIC
TRANSAMINASES AND PANCREATIC
ENZYMES ARE OFTEN ACUTELY INCREASED.
VIOLATION OF NORMAL MUCOSAL
PROTECTIVE BARRIERS PREDISPOSES THE
VICTIM TO BACTEREMIA AND PULMONARY
INFECTIONS
Management
PRE-HOSPITAL MANAGEMENT
HOSPITAL BASED MANAGEMENT
lED MANAGEMENT
lPICU MANAGEMENT
◩CARDIORESPIRATORY
◩NEUROLOGICAL
◩HYPOTHERMIA
◩OTHER
Pre hospital management
ABC’s
Initiation of ventilation is the only way to
interrupt the submersion time
C-Spine control, backboard
IV, O2, monitor, pulse ox
Correction of acidosis by sodium
bicarbonate
NO HEIMLICH
Passive Rewarming
Rapid Transport
All near drowning victims need
evaluation at a medical facility
Hospital management
OBSERVATION 6-8 HRS
MONITORING-VITALS
REPEAT RESPIRATORY SYSTEM
EXAMINATION AND NEUROLOGICAL
STATUS
If SYMPTOMS DECREASE AND
SAO2 ACHIEVED VICTIM CAN BE
DISCHARGED
Hospital management
Management in ED
◩ABC’s, with C-spine control
◩IV, O2, Monitor, Pulse Ox
◩CXR
◩ABGs
◩Electrolytes
◩Trauma workup
Acc to GCS
GCS >12
◩Oxygen to keep sat >
95%
◩Observe 4-6 hours
◩Chest signs absent
◩Saturation normal
◩Discharge home
◩No investigation
needed
GCS>12
◩Oxygen to keep sat >
95%
◩Observe 4-6 hours
◩Chest signs
present
◩Requires oxygen
◩Deteriorates
◩Admit to Monitored
bed
Acc to GCS
GCS <13
◩High flow oxygen
◩Intubation for low PaO2
◩CXR, Labs
◩Continuous cardiac monitoring
◩Frequent reassessments
Cardiopulmonary
resuscitationETT INTUBATION
ADEQUATE OXYGENATION
FLUID RESUSCITATION-ISOTONIC FLUIDS
INOTROPIC AGENTS SHOULD BE
JUDICIOUSLY USED
PERSISTENT CARDIOPULMONARY
ARREST ON ARRIVAL ALONG WITH
‱ APNEA.
‱ ABSENCE OF PUPILLARY RESPONSES.
‱ HYPERGLYCEMIA.
‱ SUBMERSION DURATIONS >10 MIN.
‱ FAILURE OF RESPONSE TO CPR GIVEN
FOR 25 MIN.
NEUROLOGICAL MANAGEMENT
HYPOTHERMIA
Passive External
Active External
Active Internal
◩IV
◩Vent
◩NG/Bladder/Peritoneal
OTHER MANAGEMENT
ISSUES
ACUTE RENAL
FAILURE :
‱ DIURETICS,
‱ FLUID RESTRICTION.
‱ DIALYSIS IF
REQUIRED
PROFUSE BLOODY
DIARRHEA AND
MUCOSAL SLOUGHING
:
‱ BOWEL REST.
‱ NASOGASTRIC
SUCTION.
FEVER
◩COMMON IN 50%
CASES WITHIN 24
HRS
◩PROPHYLACTIC
ANTIBIOTICS ARE
RECOMMENDED
ONLY IF
PNEUMONIA IS
SUSPECTED
Drowning
Drowning
Drowning

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Drowning

  • 2. WHAT IS DROWNING? “The process of experiencing respiratory impairment from submersion/immersion in liquid ” It is a form of asphyxia. Caused due to aspiration of fluids
  • 3. Epidemiology Incidence of drowning- 2.5 lakhs to 3.5 lakhs every year India & China account for 43% deaths. Males>Females 1-4 yrs & 15-19 yrs(M.C) Drowning second most common accidental cause of death in children
  • 4. Causes of Drowning CHILDREN YOUNGER THAN 1 YR : ◩‱ BATHTUB(71%) ◩ ‱ HOUSEHOLD BUCKETS(16%) CHILDREN 1-4 YR : ◩ ‱ POOL ◩ ‱ IRRIGATION DITCHES ◩ ‱ NEARBY PONDS & RIVERS. ï‚—â€ą SCHOOL AGE CHILDREN ◩‱ SWIMMING OR BOATING ACTIVITIES. ◩‱ NATURAL WATER RESERVOIRS : LAKES, PONDS, RIVERS, CANALS.
  • 5. Causes of Drowning ADOLESCENT l M : F = 10 : 1. ◩(LIKELY DUE TO GREATER RISK TAKING & ALCOHOL USE.) ◩70 % DEATHS DUE TO DROWNING IN NATURAL WATER RESERVOIRS. ï‚—â€ą UNDERLYING CONDITIONS ◩ EPILEPSY ◩ VENTRICULAR ARRHYTHMIAS ◩ ALCOHOL USE ◩ WATER SPORTS & RECREATIONAL ACTIVITIES
  • 6. Classification of drowning. According to type of water ◩Fresh-water drowning: Lakes,pools,rivers ◩Salt-water drowning: seas (about 3% saline) ï‚—â€ą According to water temperature ◩Warm-water drowning: temperatures of > 20° C lCold-water drowning: temperatures of <20° C ◩Very-cold-water drowning: temperatures of <5ÂșC
  • 7. TYPES OF DROWNING FOUR TYPES:- 1.WET DROWNING 2.DRY DROWNING 3.SECONDARY/NEAR DROWNING 4.IMMERSION SYNDROME
  • 8. WET DROWNING Water is inhaled into lungs Victim has severe chest pain On resuscitation: no pleasant recollections Death occurs due to Cardiac arrest or ventricular fibrillation
  • 9. Dry drowning Water does not enter lungs On resuscitation : panoramic views of past life. Death occurs by immediate sustained laryngeal spasm due to inrush of water into nasopharynx and larynx
  • 10. Secondary drowning (near drowning) Refers to a submerged victim who is resuscitated and survives for 24 hours Death occurs ( from 1/2 h to several days) by cerebral anoxia & irreversible brain damage
  • 11. Immersion Syndrome Death occurs by cardiac arrest caused by vagal inhibition (cold water stimulating nerve endings & water striking epigastrium & Alcohol induce such effect) Mostly seen in suicide cases.
  • 12.
  • 13. Stages of drowning Stage of surprise(5-10 secs) Stage of 1st resp.arrest.(1-2 min) Stage of deep respiration(1 min) Stage of 2nd resp.arrest (1 min) Stage of terminal gasp.
  • 14.
  • 15. lFRESH WATER DROWNING Water absorbed into circulation Surfactant washout Alveolar cell damage Chemical pneumonitis, pulmonary edema Hypervolemia Hyponatremia Hemodilution Hemolysis Hyperkalemia lSALT WATER DROWNING Hypertonic Protein rich effusion into alveoli Surfactant damage, alveolar basement membrane damage Alveolar cell damage Chemical pneumonitis, pulmonary edema Hypovolemia Hypernatremia Hemoconcentration Hypotonic
  • 16. Cause of death Asphyxia: due to airway obstruction Hypothermia Ventricular fibrillation: due to anoxia & disturbed sodium-potassium ratio Laryngeal spasm Vagal inhibition: in icy water due to high emotion & unexpected immersion Exhaustion Injury
  • 17. SYMPTOMS AND SIGNS 75% of kids who develop symptoms do so within 7 hours of event Coma to agitated alertness Cyanosis, coughing, and the production of frothy white sputum Tachypnea, tachycardia Low-grade fever Rales, rhonchi & less often wheezes Signs of associated trauma to the head and neck should be sought History is the most important
  • 18. EFFECT ON VARIOUS SYSTEMS LUNG : ◩ARDS. ◩PULMONARY OEDEMA HEART : ◩MYOCARDIAL DYSFUNCTION. ◩ARTERIAL HYPOTENSION. ◩DECREASED CARDIAC OUTPUT. ◩ARRHYTHMIAS & CARDIAC INFARCTION.  RENAL : ◩ACUTE TUBULAR NECROSIS ◩CORTICAL NECROSIS ◩RENAL FAILURE.
  • 19. VASCULAR ENDOTHELIAL INJURY MAY INITIATE ◩DIC, ◩HEMOLYSIS, ◩THROMBOCYTOPENIA.  GASTROINTESTINAL DAMAGE : ◩BLOODY DIARRHEA WITH MUCOSAL SLOUGHING. ◩SERUM LEVELS OF HEPATIC TRANSAMINASES AND PANCREATIC ENZYMES ARE OFTEN ACUTELY INCREASED. VIOLATION OF NORMAL MUCOSAL PROTECTIVE BARRIERS PREDISPOSES THE VICTIM TO BACTEREMIA AND PULMONARY INFECTIONS
  • 20. Management PRE-HOSPITAL MANAGEMENT HOSPITAL BASED MANAGEMENT lED MANAGEMENT lPICU MANAGEMENT ◩CARDIORESPIRATORY ◩NEUROLOGICAL ◩HYPOTHERMIA ◩OTHER
  • 21. Pre hospital management ABC’s Initiation of ventilation is the only way to interrupt the submersion time C-Spine control, backboard IV, O2, monitor, pulse ox Correction of acidosis by sodium bicarbonate NO HEIMLICH Passive Rewarming Rapid Transport All near drowning victims need evaluation at a medical facility
  • 22.
  • 23. Hospital management OBSERVATION 6-8 HRS MONITORING-VITALS REPEAT RESPIRATORY SYSTEM EXAMINATION AND NEUROLOGICAL STATUS If SYMPTOMS DECREASE AND SAO2 ACHIEVED VICTIM CAN BE DISCHARGED
  • 24. Hospital management Management in ED ◩ABC’s, with C-spine control ◩IV, O2, Monitor, Pulse Ox ◩CXR ◩ABGs ◩Electrolytes ◩Trauma workup
  • 25. Acc to GCS GCS >12 ◩Oxygen to keep sat > 95% ◩Observe 4-6 hours ◩Chest signs absent ◩Saturation normal ◩Discharge home ◩No investigation needed GCS>12 ◩Oxygen to keep sat > 95% ◩Observe 4-6 hours ◩Chest signs present ◩Requires oxygen ◩Deteriorates ◩Admit to Monitored bed
  • 26. Acc to GCS GCS <13 ◩High flow oxygen ◩Intubation for low PaO2 ◩CXR, Labs ◩Continuous cardiac monitoring ◩Frequent reassessments
  • 27. Cardiopulmonary resuscitationETT INTUBATION ADEQUATE OXYGENATION FLUID RESUSCITATION-ISOTONIC FLUIDS INOTROPIC AGENTS SHOULD BE JUDICIOUSLY USED PERSISTENT CARDIOPULMONARY ARREST ON ARRIVAL ALONG WITH ‱ APNEA. ‱ ABSENCE OF PUPILLARY RESPONSES. ‱ HYPERGLYCEMIA. ‱ SUBMERSION DURATIONS >10 MIN. ‱ FAILURE OF RESPONSE TO CPR GIVEN FOR 25 MIN.
  • 29. HYPOTHERMIA Passive External Active External Active Internal ◩IV ◩Vent ◩NG/Bladder/Peritoneal
  • 30. OTHER MANAGEMENT ISSUES ACUTE RENAL FAILURE : ‱ DIURETICS, ‱ FLUID RESTRICTION. ‱ DIALYSIS IF REQUIRED PROFUSE BLOODY DIARRHEA AND MUCOSAL SLOUGHING : ‱ BOWEL REST. ‱ NASOGASTRIC SUCTION. FEVER ◩COMMON IN 50% CASES WITHIN 24 HRS ◩PROPHYLACTIC ANTIBIOTICS ARE RECOMMENDED ONLY IF PNEUMONIA IS SUSPECTED